NR 341/ NR341 COMPLEX ADULT HEALTH EXAM 1 LATEST 2023-2024 QUIZBANK & EXAM QUESTIONS AND CORRECT ANSWERS|AGRADE

Exam 1

6:ekg

4:abg

A patient with multiple draining wounds is admitted for hypovolemia. What would be the most accurate

way for the nurse to evaluate fluid balance?

a. Skin turgor

b. Daily weight

c. Urine output

d. Edema presence

A patient receives 3% NaCl solution for correction of hyponatremia. Which assessment is most important

for the nurse to monitor while the patient is receiving this infusion?

a. Lung sounds

b. Urinary output

c. Peripheral pulses

d. Peripheral edema

Following a thyroidectomy, a patient reports “a tingling feeling around my mouth.” Which assessment

should the nurse complete first?

a. Verify the serum potassium level.

b. Test for presence of Chvostek’s sign.

c. Observe for blood on the neck dressing.

d. Confirm a prescription for thyroid replacement.

The nurse has just auscultated coarse crackles bilaterally on a patient with a tracheostomy tube in place.

If the patient is unsuccessful in coughing up secretions, what action should the nurse take?

a. Encourage increased incentive spirometer use.

b. Encourage the patient to increase oral fluid intake.

c. Put on sterile gloves and use a sterile catheter to suction.

d. Preoxygenate the patient for 3 minutes before suctioning.

Which action should the nurse take first when a patient develops epistaxis?

a. Pack the affected nare tightly with an epistaxis balloon.

b. Apply squeezing pressure to the nostrils for 10 minutes.

c. Obtain silver nitrate that may be needed for cauterization.

d. Instill a vasoconstrictor medication into the affected nare.

A patient arrives in the emergency department with a possible nasal fracture after being hit by a

baseball. Which finding by the nurse is most important to report to the health care provider?

a. Clear nasal drainage

b. Report of nasal pain

c. Bilateral nose swelling and bruising

d. Inability to breathe through the nose

When assessing a patient with a sore throat, the nurse notes anterior cervical lymph node swelling, a

temperature of 101.6° F (38.7° C), and yellow patches on the tonsils. Which action will the nurse

anticipate taking?

a. Teach the patient about the use of expectorants.

b. Use a swab to obtain a sample for a rapid strep antigen test.

c. Discuss the need to rinse the mouth out after using any inhalers.

d. Teach the patient to avoid nonsteroidal anti-inflammatory drugs (NSAIDs).

A patient with respiratory failure has a respiratory rate of 6 breaths/min and an oxygen saturation (SpO2)

of 78%. The patient is increasingly lethargic. Which intervention will the nurse anticipate?

a. Administration of 100% O2 by non-rebreather mask

b. Endotracheal intubation and positive pressure ventilation

c. Insertion of a mini-tracheostomy with frequent suctioning

The oxygen saturation (SpO2) for a patient with left lower lobe pneumonia is 90%. The patient has

wheezes and a weak cough effort. Which action should the nurse take?

a. Position the patient on the left side.

b. Assist the patient with staged coughing.

c. Place a humidifier in the patient’s room.

d. Schedule a 4-hour rest period for the patient.

A nurse is caring for a patient with right lower lobe pneumonia who is obese. Which position will provide

the best gas exchange?

a. On the left side

b. On the right side

c. In the tripod position

d. In the high-Fowler’s position

A nurse is caring for a patient who is orally intubated and receiving mechanical ventilation. To decrease

the risk for ventilator-associated pneumonia, which action will the nurse include in the plan of care?

a. Elevate head of bed to 30 to 45 degrees.

b. Give enteral feedings at no more than 10 mL/hr.

c. Suction the endotracheal tube every 2 to 4 hours.

d. Limit the use of positive end-expiratory pressure.

A patient admitted with acute respiratory failure has ineffective airway clearance from thick secretions.

Which nursing intervention would specifically address this patient problem?

a. Encourage use of the incentive spirometer.

b. Offer the patient fluids at frequent intervals.

c. Teach the patient the importance of ambulation.

d. Titrate oxygen level to keep O2 saturation above 93%.

The nurse observes a new onset of agitation and confusion in a patient with chronic obstructive

pulmonary disease (COPD). Which action should the nurse take first?

a. Observe for facial symmetry.

b. Notify the health care provider.

c. Attempt to calm and reorient the patient.

d. Assess oxygenation using pulse oximetry.

The nurse is admitting a patient who has chest pain. Which assessment data suggest that the pain is

from an acute myocardial infarction?

a. The pain increases with deep breathing.

b. The pain has lasted longer than 30 minutes.

c. The pain is relieved after the patient takes nitroglycerin.

d. The pain is reproducible when the patient raises the arms.

Which information from a patient helps the nurse confirm the previous diagnosis of chronic stable

angina?

a. “The pain wakes me up at night.”

b. “The pain is level 3 to 5 (0 to 10 scale).”

c. “The pain has gotten worse over the last week.”

d. “The pain goes away after a nitroglycerin tablet.”

Which patient statement indicates that the nurse’s teaching about sublingual nitroglycerin (Nitrostat) has

been effective?

a. “I can expect nausea as a side effect of nitroglycerin.”

b. “I should only take nitroglycerin when I have chest pain.”

c. “Nitroglycerin helps prevent a clot from forming and blocking blood flow to my heart.”

d. “I will call an ambulance if I have pain after taking 3 nitroglycerin 5 minutes apart.”

A patient who has had chest pain for several hours is admitted with a diagnosis of rule out acute

myocardial infarction (AMI). Which laboratory test is most specific for the nurse to monitor in

determining whether the patient has had an AMI?

a. Myoglobin

b. Homocysteine

c. C-reactive protein

d. Cardiac-specific troponin

Heparin is ordered for a patient with a non–ST-segment-elevation myocardial infarction (NSTEMI). How

should the nurse explain the purpose of the heparin to the patient?

a. “Heparin enhances platelet aggregation at the plaque site.”

b. “Heparin decreases the size of the coronary artery plaque.”

c. “Heparin prevents the development of new clots in the coronary arteries.”

d. “Heparin dissolves clots that are blocking blood flow in the coronary arteries.”

Which action will the nurse take to evaluate the effectiveness of IV nitroglycerin for a patient with a

myocardial infarction (MI)?

a. Monitor heart rate.

b. Ask about chest pain.

c. Check blood pressure.

d. Observe for dysrhythmias.

Diltiazem (Cardizem) is prescribed for a patient with newly diagnosed Prinzmetal’s (variant) angina.

Which action of diltiazem is accurate for the nurse to include in the teaching plan?

a. Reduces heart palpitations.

b. Prevents coronary artery plaque.

c. Decreases coronary artery spasms.

d. Increases contractile force of the heart.

A patient recovering from a myocardial infarction (MI) develops chest pain on day 3 that increases when

taking a deep breath and is relieved by leaning forward. Which action should the nurse take as focused

follow-up on this symptom?

a. Assess both feet for pedal edema.

b. Palpate the radial pulses bilaterally.

c. Auscultate for a pericardial friction rub.

d. Check the heart monitor for dysrhythmias.

A patient who is recovering from an acute myocardial infarction (AMI) asks the nurse about safely

resuming sexual intercourse. Which response by the nurse is best?

a. “Most patients are able to enjoy intercourse without any complications.”

b. “Sexual activity uses about as much energy as climbing two flights of stairs.”

c. “The doctor will provide sexual guidelines when your heart is strong enough.”

d. “Holding and cuddling are good ways to maintain intimacy after a heart attack.”

A patient who has recently started taking pravastatin (Pravachol) and niacin reports several symptoms to

the nurse. Which information is most important to communicate to the health care provider?

a. Generalized muscle aches and pains

b. Dizziness with rapid position changes

c. Nausea when taking the drugs before meals

d. Flushing and pruritus after taking the drugs

A patient had a non–ST-segment-elevation myocardial infarction (NSTEMI) 3 days ago. Which nursing

intervention is appropriate for the registered nurse (RN) to delegate to an experienced licensed

practical/vocational nurse (LPN/VN)?

a. Reinforcement of teaching about the prescribed medications

b. Evaluation of the patient’s response to walking in the hallway

c. Completion of the referral form for a home health nurse follow-up

d. Education of the patient about the pathophysiology of heart disease

The nurse is caring for a patient who is receiving IV furosemide (Lasix) and morphine for the treatment of

acute decompensated heart failure (ADHF) with severe orthopnea. Which clinical finding is the best

indicator that the treatment has been effective?

a. Weight loss of 2 lb in 24 hours

b. Hourly urine output greater than 60 mL

c. Reduced dyspnea with the head of bed at 30 degrees

d. Patient denies experiencing chest pain or chest pressure

A patient who has chronic heart failure tells the nurse, “I was fine when I went to bed, but I woke up in

the middle of the night feeling like I was suffocating!” How should the nurse document this finding?

a. Orthopnea

b. Pulsus alternans

c. Paroxysmal nocturnal dyspnea

d. Acute bilateral pleural effusion

Which diagnostic test will be most useful to the nurse in determining whether a patient admitted with

acute shortness of breath has heart failure?

a. Serum troponin

b. Arterial blood gases

c. B-type natriuretic peptide

d. 12-lead electrocardiogram

A patient who has chronic heart failure is admitted to the emergency department with severe dyspnea

and a dry, hacking cough. Which action should the nurse take first?

a. Auscultate the abdomen.

b. Check the capillary refill.

c. Auscultate the breath sounds.

d. Ask about the patient’s allergies.

A patient with chronic heart failure who is taking a diuretic and an angiotensin-converting enzyme (ACE)

inhibitor and who is on a low-sodium diet tells the home health nurse about a 5-lb weight gain in the

past 3 days. What is the nurse’s priority action?

a. Teach the patient about restricting dietary sodium.

b. Assess the patient for manifestations of acute heart failure.

c. Ask the patient about the use of the prescribed medications.

d. Have the patient recall the dietary intake for the past 3 days.

After receiving change-of-shift report on four patients admitted to a heart failure unit, which patient

should the nurse assess first?

a. A patient who reported dizziness after receiving the first dose of captopril.

b. A patient who has new-onset confusion and restlessness and cool, clammy skin.

c. A patient who is receiving oxygen and has crackles bilaterally in the lung bases.

d. A patient who is receiving IV nesiritide (Natrecor), with a blood pressure of 100/62.

What should the nurse measure to determine whether there is a delay in impulse conduction through

the patient’s ventricles?

a. P wave

b. Q wave

c. PR interval

d. QRS complex

The nurse needs to quickly estimate the heart rate for a patient with a regular heart rhythm. Which

method will be fastest to use?

a. Count the number of large squares in the R-R interval and divide by 300.

b. Print a 1-minute electrocardiogram (ECG) strip and count the number of QRS complexes.

c. Use the 3-second markers to count the number of QRS complexes in 6 seconds and multiply by 10.

d. Calculate the number of small squares between one QRS complex and the next and divide into 1500.

A patient has a junctional escape rhythm on the monitor. What heart rate should the nurse expect the

patient to have?

a. 15 to 20

b. 20 to 40

c. 40 to 60

d. 60 to 100

A patient has a sinus rhythm and a heart rate of 72 beats/min. The nurse determines that the PR interval

is 0.24 seconds. What action should the nurse take?

a. Notify the health care provider immediately.

b. Document the finding and monitor the patient.

c. Give atropine per agency dysrhythmia protocol.

d. Prepare the patient for temporary pacemaker insertion.

A patient who was admitted with a myocardial infarction has a 45-second episode of ventricular

tachycardia, then converts to sinus rhythm with a heart rate of 98 beats/min. Which action should the

nurse take next?

a. Immediately notify the health care provider.

b. Document the rhythm and continue to monitor the patient.

c. Prepare for synchronized cardioversion per agency protocol.

d. Prepare to give IV amiodarone per agency dysrhythmia protocol.

After the nurse gives IV atropine to a patient with symptomatic type 1, second-degree atrioventricular

(AV) block, which finding indicates that the drug has been effective?

a. Increase in the patient’s heart rate

b. Increase in strength of peripheral pulses

c. Decrease in premature atrial contractions

d. Decrease in premature ventricular contractions

A patient with dilated cardiomyopathy has new onset atrial fibrillation that has been unresponsive to

drug therapy for several days. What topic should the nurse plan to include in patient teaching?

a. Anticoagulant therapy

b. Permanent pacemakers

c. Emergency cardioversion

A patient on the telemetry unit develops atrial flutter, rate 150, with associated dyspnea and chest pain.

Which action that is included in the agency dysrhythmia protocol should the nurse do first?

a. Obtain a 12-lead electrocardiogram (ECG).

b. Notify the health care provider of the change in rhythm.

c. Give supplemental O2 at 2 to 3 L/min via nasal cannula.

d. Assess the patient’s blood pressure and discomfort level.

A patient is apneic and has no palpable pulses. The heart monitor shows sinus tachycardia, rate 132.

What action should the nurse take next?

a. Perform synchronized cardioversion.

b. Start cardiopulmonary resuscitation (CPR).

c. Give atropine per agency dysrhythmia protocol.

d. Apply supplemental O2 via non-rebreather mask.

Which action will the nurse include in the plan of care for a patient who was admitted with syncopal

episodes of unknown origin?

a. Explain the association between dysrhythmias and syncope.

b. Instruct the patient to call for assistance before getting out of bed.

c. Teach the patient about the need to avoid caffeine and other stimulants.

d. Tell the patient about the benefits of implantable cardioverter-defibrillators.

nr 341 syllabus
nr 341 exam 1 chamberlain
nr 341 proctored exam
nr 341 quiz 5
nr 341 exam 1 study guide
nr 341 exam 2
chamberlain complex exam 2
nr 341 quiz 4

Rapid Response

– prevents code
– s/s of septic shock

Advance Directive

– have before admission
– NO SLOW CODES
– right to say no

Visitation

– based on the needs of the patient
– hospital will never restrict visitation

Brain Death

perfusion and oxygenation until organ transplant

Respiratory Failure

– decreased pH
– increased CO2
– decreased PaO2

Intubation

– sedate
– paralyze
– auscultate
– chest xray
– fall and rise bilateral
– CO2 detector – gold is good

ARDS

– decreased Pa02 despite increasing FaO2
– injury to lungs
– tachypnic and tachycardic
– respiratory acidosis
– decreased compliance
– INCREASED PEEP

ARDS: Intervention

– prone (maintain airway)
– elevate HOB 30-45 degrees

VAP Bundle

– HOB 30-45 degrees
– Oral care w/ Chlorhexidine
– DVT Care – Heparin and Lovenox
– PPI’s prevent PUD
– Sedation Vacation
– Drain condensation away from patient

What should you do after 7 days on a Ventilator?

Trach them

Terminal Weaning

– DNR
– take them off ventilator to die
– COMFORT IS KEY, NO PAIN (benzos, pain meds)

American Association of Critical Care Nurses: Focus of the Group

– assists NURSES to attain knowledge and influence
– healthy work environment initiative

Highest setting you can put a Nasal Cannula?

6 L, humidify at 4L

Patient is on 90% Fi02 (10L), Respiratory Distress…What do I do?

INTUBATE

Overdose on CNS Depressant?

– respiratory depression
– respiratory acidosis
– hypoventilation

Panic Attack: Respiratory

– respiratory alkalosis
– hyperventilation
– tingling, numbness in fingers (decreased perfusion)

What is a sign of decreased Perfusion?

cyanosis

ARDS: Chest Xray?

– broken glass
– white out

What does PEEP increase?

PaO2

Ready to wean?

– PaO2 >60 and FiO2 <.4
– PEEP <5-8 cm
– PaO2/FiO2 should be 150-200

Vented patient is Asystole, don’t know if he has DNR?

stop the code

High Pressure Alarm

– biting the tube
– mucus plug
– kinked tube
if you can’t figure it out, bag patient and call an RT

Low Pressure Alarm

something is disconnected

Prepare you patient for weaning

tell patient breathing will be hard

Weaning is not working

– RR increases
– HR increases
– O2 decreases
– low hemoglobins will not be successful

Patient is Tachy (150), hypotensive, signs of shock, heart failure…

– vagel maneuver first
– synchronized cardioversion

Patient is brady (35), hypotensive, diaphoresis, dizzy and SOB

transcutaneous pacing

Anxious on Ventilator

– sedate
– neuromuscular block (SEDATE FIRST)
– pressure ulcers are common as well as corneal abrasions (can’t close eyes).

RASS

– agitation and sedation
– tells you if you need a neuro block
– 4 needs a neuro block
– 2 is typical

ALOC

– restless and irritable (WHY)
– typically not adequately oxygenated

Suction

– only when indicated
– hyperoxygenate before

Common Central Line Injury

pneumothorax

What is common to see with weaning?

– tachy
– agitated
– using accessory muscles

Patient w/ tachypnea, restlessness, getting worse, hypoxia…

ARDS

What is the first thing to do when patient alarms go off?

assess the patient

Coding Protocol

100 compressions – 2 minutes – check pulse

Asystole

– start meds
– epi (1:10,000, 1 mg)

Heparin: preventive/ Therapeutic/ use

Preventive:
-SubQ (SC)
-5000 units BID/ TID

Therapeutic:
-IV
-weight based
-units/Kg/ hr
-w/ loading dose

Use on Pt:
-(+) PE
-(+) VTE/ DVT
-off warfarin for impending procedure
ex: mechanical valve

Heparin: monitor Pt for

-platelets
-clot times (Q4H)
aptt/ ptt/ anti-Xa
-Hgb
baseline & drop

H.I.T: Heparin induced thrombocytopoenia

Enoxaparin: preventive/ therapeutic/ use

Preventive:
-SC 30 to 40 mg/ daily

Therapeutic:
-SC
-mg/Kg
-1.5 mg/ Kg/ daily or 1 mg BID

Ulcers:

Use:
-PPI
-H2 blockers

*PPI increase RF for C-diff*

Bundle of Care

A. Assessment of pain (CPOT)
B. SAT/ SBT (spontaneous awakening trial/ spontaneous breathing trial)
C. Choice of analgesics & sedation
D. Delirium (ICU psychosis)
E. early mobility
F. Family involvement

A: Assessment of pain

-regional anesthesia/ nonopioid adjuncts
-analgesia-based sedation tech. w/ fentanyl

B: Both SAT & SBT

-Daily linked SAT/ SBT
-multidisciplinary coordination of care
-faster liberation from mech. vent.

C: Choice of sedation

-targeted light sedation when sedation nec.
-avoidance of benzo’s
-dexmedetomidine(precedex) if high delirium risk/ cardiac surg./MV weaning

D: Delirium monitoring & management

-routine CAM-ICU assessements
-nonpharmacologic interventions, including sleep hygiene
-precedex or antipsychotic if hyperactive Sx

E: Early mobility & exercise

-PT/ OT assessment
-coordinate w/ SAT or periods of no sedation
-progress thu ROM, sitting, standing, walking, ADLs

F: family engagement & empowerment

-reorientation, provision of emotional and verbal support
-cognitive stimulation, participation in mobilization
-participation in multi-disciplinary rounds/care

ICU psychosis (Delirium)

acute confusion after 2 to 3 days in the intensive care unit

RF:
-Hx of delirium
->70 y.o.
-environmental
-critical illness/ acute/ major surg.
-ventilator
-meds
-ICU > 3 days
-Hx of dementia
-inadequate pain control
Tx:
-therapeutic comms
*not reversible 100% of time*
-Sitter
-surveillance

RF meds for delirium:

-psychotropic
-benzo
-opioids
also caused by inadequate pain control

Delirium Tx:

Tx:
-therapeutic comms
*not reversible 100% of time*
-Sitter
-surveillance
-Haldol preferred
-all equipment to maintain senses (glasses/ hearing aids)
-what caused the delirium: use of pshyotropics/ benzo/ opioids/ environmental/ family/ all RF listed prior.

Haldol side effects

-anti-cholinergic
-dry eyes
-dry mucus membranes
-EPS (Cogentin)
-drowsiness

Delirium prevention

Familiarization
-person/ place/ time
-current events
-items
-less environmental noise (TV/ machines)
-CAM

RASS

Richmond Agitation Sedation Scale
– = sedation
+ = agitation

*-5 no response*
-4 deep sedation
-3 moderate sedation (any movement to voice)
-2 light sedation (<10 secs w/ eye contact)
-1 drowsy (>10 secs w/ eye contact)
*0 Alert and calm* baseline
+1 Restless
+2 agitated
+3 very agitated
*+4 combative*- violent/ danger to staff

Goal of sedation on RASS

– (-)1 to (-) 2
-usually titration orders

If Pt is +4 what do we admin:

-NO benzo’s
-Admin Haldol (anti-cholinergic)
dry mouth
increase QT
EPS: admin cogentin

Last result for 4+ RASS (last resort)

Paralytics
-vecuronium
-succinycholine
sedation/ analgesia/ amnesia

Train of 4

*Goal of TOF is about 2 twitches: ulnar/ facial/ posterior tibial nerves
For paralytics:
0-4
Peripheral nerve stimulator
-0- total block (too much)
-1 to 2- goal
-4- no blockage (too little)
misc:
-BIS (Bispectral index/ EEG)
-monitor sedation
-RASS = -5, therefore RASS not used if paralyzed
-CPOT= also masked by paralytic

Continuous sedation monitored by:

BIS

Paralytic monitored by:

Train of 4

During paralytic use analgesics for pain monitored by:

-no monitor
-constant sedation and pain meds admin

Most delivered O2

BVM

RSI (Rapid Sequence Intubation)

Sedative
-Ketamine 1-2mg/kg
-Propofol 2mg/kg
Etomidate 0.3mg/kg
-Midazolam 1mg SIVP

Neuromuscular Blocker(paralytic)
-Vocuronium (ROC) 1mg/kg Adult – 100mg (Give before sedative)
-Succinylcholine 1mg/kg Adult 100mg (Give after sedative)

Nursing responsibilites:
-no more than 30 secs during attempt
-ventilate for 3-5 mins before reattempt
-Equip gather
-hyper O2
-sedate (etomidate or precedex)
-paralytic
-tube placement & check w/ chest expansion bilateral and auscultation
-CO2 detector- Gold is good
-Chest x-ray is still the GOLD standard

Use of precedex

-no respiratory depression

Intubation indications

Unable to swallow
Patient can not ventilate
Inhalation burns
Anaphylaxis
Apnea
Obstruction
Respiratory failure
Trauma
TBI
Post surgery
non-patent airway

Why do we paralyze

Decrease O2 demands
-ARDS
Prevent Pt-vent dysynchrony
-VDR vents
-Bi Vents
Prevent shivering in hypothermia Pt
-shivering increases O2 demand
-raises Pt temp.
Open chest

checklist for chemical paralysis

-adequately sedated first before paralytic admin
-must have anxiolytic drip that has amnesic properties
-must have analgesic drip infusing
-must have lubrication for eyes/ eye bubbles

Propofol (Diprivan)

class: general anesthetic
Indication: anesthesia, induction, sedation
Action: hypnotic, produces amnesia

Nursing Considerations:
– use cautiously with CVD, lipid disorder, increased ICP
– can cause apnea, bradycardia, *hypotension*
– burning and pain at insertion site
– *can turn urine green*
– assess respiratory status and hemodynamics
– maintain patent airway
– assess level of sedation

-5 to 10 mcg/kg/min
-use when ventilated or qualified MD or nurse anesthetist
-*do not use if allergic to egg/soybean*

Precedex (dexmedetomidine)

MOA: selective alpha2- adrenoreceptor agonist
-Tx of anxiety and sedation/ pain

Nursing considerations:
-monitor HP/ BP
-hypotension/ brady

Admin: bolus, then cont. infusion

Haloperidol

-anti-cholinergic
-MOA
-decrease cerebral cortex function
A/E:
-*extend QT wave*
*ADD MORE*

CPOT

critical care pain observation tool
-scale of 0 thru 8 used to quantify non-verbal pain
-observe minimum of :01
-greater than 3 indicates pain.
1. Facial: Grimacing-2
2. Body movements: restelessness/ agitation-2
3a. Ventilator compliance (intubated):fighting vent-2
3b. Vocalization (extubated):cyring out/sobbing-2
4. Muscle tension:very tense/ rigid-2

Propofol MOA

GABA agonist

Propofol class

Sedative/Hypnotic

Benzodiazepines antidote

Flumazenil (Romazicon)

Opioid antidote

Naloxone (Narcan)

Versed (midazolam): class

Benzodiazepine

Versed MOA

-CNS depressant
-Produces anterograde amnesia, then sedation
-Stops and prevents SZs
flumazenil antidote

advanced directive (living will)

details the conditions under which life support measures should be used
-is an outside of hospital document
-MPOA can over-ride LW
-have spokesperson established for relating info

DNR (do not resuscitate)

an order that tells medical professionals not to perform CPR
-document that is an order from hospital
-from physician
-if phone order- 2 nurse verification (witness)

Intubation duration:

Two weeks max, then tracheostomy
-ease
-swelling
-less infection
-no gag reflex

Nutrition requirements

-protein requirements increase
-admin w/in 24 hours of admittance

Types of nutrition:

Enteral
Parenteral

Parentaral nutrition

Total parentaral nutrition (TPN)
-electrolytes
-amino acids
-glucose (50-70% for CPN & 10% for PPN)
-lipids (not to Pt w/ increased cholesterol)
*check BG/BS on Pt*=> infection

TPN infected central line

-area is reddened
-increase WBC
cannot infiltrate on central line
-if stopped, hang D-10 (dextrose)

Enteral nutrition

giving nutrients into the gastro-intestinal tract through a feeding tube
-depends on illness, specially formulated for each by a dietician
-does Pt have a “working gut”?
-decreased cost
-decreased risk for secondary infection

Working gut

-flora
-peristalsis
-mucous layer

If Pt does not have working gut for enteral nutrition

-can cause breakdown and secondary infections
-decrease immunity of Pt

HOB for enteral feeding

-30 degrees or higher for aspiration
*if hypotensive 10 degrees or less*

Duodenal tube

less chance of aspiration (greatly reduced)
-aspiration of contents: yellow = duodenum/ green = stomach
-confirm w/ chest x-ray or KUB
-pH only works if NPO and no h2 blocker/ PPI
-can use Raglen to advance duo-tube

How to test for nutrition efficiency

pre-albumin
16-30 value

ABG ranges

pH 7.35-7.45
PaCO2 35-45
HCO3 22-26
PaO2 80-100

Big box method

measure the R-R interval, count the number of big boxes between & divide into 300

Small Box method (ECG)

count number of small boxes between a R-R and divide into 1500 (one min =1500 small boxes).

Six second method

Count the number of QRS complexes in a 6 second strip and multiply by 10

P-wave
0.12-0.20

atrial depolarization (contraction)

If P-wave is present

-Sinus rhythm
-not present= not sinus

PR interval

0.12-0.20 seconds (3-5 small boxes)
-SA node firing => vent. contraction (purkinje fibers)
-delay of AV node to fill ventricles
>0.20 = 1st degree heart block

PR interval

extended PR interval
-1st degree block

QRS complex

0.06 -0.10
< 0.10 or 2.5 small boxes
> 0.10 = delay in ventricle
-ventricular depolarization (contraction) and atrial repolarization

QT interval

Ventricular depolarization(contraction) and repolarization(relaxation).
Normal interval is 0.32 to 0.44 seconds.(< 0.45)
-lethal > 0.45
>0.50 = 911
can be extended by Haldol, zofran, and lots of other drugs(anti-d.rythmics)
-know Pt baseline QT

T-wave can be altered by

K+ (potassium)
-increased T-wave = increase of K+

ECG interpretation

-HR: normal/ fast/slow
-Rhythm: normal/ fast/ slow
-P waves: present or not
-PR interval: normal/ short/ long
-QRS: normal/ narrow = atrium & wide = ventricle

SBT (to come off vent./wean)

-calm/ cooperative
-pH > 7.3
-FiO2 < 0.60
-hemodynamically stable

Termination SBT/ not tolerating

Start back up on vent
-RR > 35
-SpO2 <90
-dysrhythmias
-increase HR
-decrease BP
-accessory muscle use
-increase anxiety
-diaphoresis

Terminal weaning

withdrawal of life-sustaining therapy with the understanding that death may result, generally after a decision is made that the therapy in question is medically futile or disproportionately burdensome
-analgesia
-sedation

terminal extubation

EOL care
-DNR order on chart
-cannot extubate while on propofol
-meds dosage increased on term. Pt
-family conference
-donor network called
-orders for comfort care (stopped curative care/ cont. palliative-comfort)

Post extubation

most critical
-Pt as upright as possible (HOB)
-no turning/ no moving to chair
-> 2 hrs & stable usually no sedation for those 2 hours
-limit talking due to vocal cord strain
-NPO until swallow evaluation

Sinus tachycardia

>100 bpm

Sinus Bradycardia

<60 bpm

PVC (Premature Ventricular Contraction)

Associated with:
-electrolyte imbalances (especially K+)
-stimulants (caffeine, ETOH, energy drinks)
-stress
-hypoxia
-heart disease

-ventricular contraction
-6 in a min or more then 3 in a row = RF lethal dysrythmias = v.tach

Tx:
-underlying cause
-amiodorone/ lidocaine
-end stage/ advanced HF will have ICD (implantable cardioverter defib placed)

multifocal PVC

-varied shapes and forms of the PVCs, suggesting more than one irritable focus
-multiple foci: positie and negative

Bigeminy PVC

-every other beat PVC
-feel radial pulse and compare to apical pulse to check perfusion

Trigeminy PVC

-every two beats, then PVC
-same as bigeminy

R on T phenomenon

-PVCs land on T wave
-can send Pt into Vtach/ torsade’s de point/ v-fib

absolute refractory period

-muscle cell is depolarized so cannot respond to another stimulus

relative refractory period

-cell is repolarizing/ heart muscle is starting to relax
-if PVC falls on this part, causes Vtach/ Vfib

1st degree heart block

PR interval >0.20
e/t rheumatic feer, digoxin, beta blocker, inferior MI, Increase vagal tone

V-fib/ unstable- pulseless V-tach algorithm/ pulseless torsades

-CAB
-CPR x 2 mins- call code/ help
-D-fib
-CPR x 2mins- EPI 1mg IVpush
-D-fib
-CPR x 2mins- amiodoarone 300mg IVpush/ mag sulfate for torsades 1-2gm

Torsades de pointes QRS:

Loos like VT but twists around baseline (changes axis and amp)
-deflect up and downwards

Torsades des pointes associations

-hypomagnesemia
-hypokalemia
-hypocalcemia
-certain antiarrhythmic drugs that prolong the QT interval (procainamide)

V-tach QRS

-wide and bizarre
– > 0.12 secs

ventricular fibrillation

the rapid, irregular, and useless contractions of the ventricles
-ventricular quiver- no contraction of atria or ventricles
-no pulse
-no CO
-no BP
-lethal
Tx: D-fibrillate

Vfib associations

-MI
-untreated VT
-HF
-electric shock

Tx: ACLS
Vfib= Dfib

V-fib ACLS

-has no pulse (like pulseless v-tach)
-call for help (code cart)
-start CPR
-D-fib when available
-resume CPR and give 1st drug: Epi 1mg IV push (circ: 2 mins)
-stop CPR and rescue breathing- analyze heart rhythm and d-fib
-resume CPR w/ rescue breathing and give 2nd drug: Amiodorone 300 mg IV push

Atrial flutter

atrial fibrillation

rapid, random, ineffective contractions of the atrium

PEA

-pulseless
-electrical activity on ECG
-poor prognosis
-clinically dead

H & T’s

Asystole

-no electrical or mechanical activity
-no CO
-no BP
-no Pulse
-“flat line”

Tx: no D-fib
-CPR and EPI/ vaso

Asystole/PEA: algorithm

verify in 2 leads/ assess Pt
-ABC’s (CAB)
-CPR x 2mins (call code/ help)
-EPI 1mg IVpush every 3-5 mins
-H & T’s
-ACLS protocol, tx like asystole NO D-fib

Asystole/ PEA: H & T’s

H:
-hypoxia (give o2/ airway)
hypovolemia (Fluid via IV)
-hypothermia (warm-up)
-Hydrogen ions (bicarbonate)
-hyper/ hypo kalemia (low- can’t do much/ high-dextrose & insulin IV)

T:
-Toxins/tablets -drug O.D.
-Tamponade (tap the heart)
-Tension pneumothorax (needle decompression)
-Thrombus (PE= dead/ Cardiac= tPA)

VAP pathogenesis

ventilator associated pneumonia
-develops 48 hours or longer after intubation
-27% of Pt occurs
-secretions pass cuff to bronchioles
-predisposes to gram-negative PNA
-Pt @ risk anytime on a vent

VAP bundle for prevention

– HOB 30-45 degrees
– interrupt sedation daily to assess readiness to wean
– provide daily oral care with chlorhexidine
– provide prophylaxis for DVT and PUD(ulcers)
-get off vent ASAP
-drain tubing away from Pt
-appropriate PPE to prevent infection

VAP: how to thin secretions

-turn Pt
-mucolytic
-resp. therapy vest
-correct antibiotic administration
no use of saline bullets

VAP: suction risk

-trauma
-decreased O2 stats => d.rythmias=> PVC
-secondary infection

VAP confirmation

-> 48 hours on vent
-sputum
-new infltrates on x-ray
-new fever

Early signs of respiratory distress

-restless
-agitation
-increase RR (tachypnea)
-increase HR (tachycardia)

Common respiratory tests:

-ABG
-chest x-rays
-CT w/ contrast
-high resolution CT angiography
-SpO2
-VQ scans
-end titdal CO2 monitoring

Acid base relationship with Resp. distress

-starts as Resp. alk
-ends with Resp. acidosis

Late signs of resp. distress

-cyanosis
-mottling
-bradypnea (decreased RR)

Why ventilate

-low PaO2
-high CO2
-can’t ventilate themselves (breathe on own)

Types of resp. failure

-hypoxemic
-hypercapic

Hypoxemic resp. failure

-PaO2 < or = 60mmHg on FiO2 > 50%

Hypercapnic resp. failure

-PaCO2 > or = 50mmHg w/ pH of 7.25 or less

Ventilator modes

-AC
-PC
-APRV

Ventilator Alarms
-Low Pressure
-High Pressure
-apnea

Low: disconnection/ cuff leak
High: suction for possible secretions, kinks.
-ARDS = stiffness
-occlusion (biting/ coughing/ kinked)
Apnea: oversedation/ loss of airway

If you can’t fix ventilator alarm & problem quickly, what do you do?

-BVM/ AMBU-bag
-breathes for them, call for help
-cannot leave room until fixed

AC Mode of Ventilation

go to mode
-volume control, set to 500-600mL
-same volume w/ every breathe
-monitor press (low 20’s)
-Pt can take more breathes than what is set (set: 15, Pt takes 10 = 25 => Hyperventilation
Concern:
-Hyperventilation
-Resp. Alk.
ex:
Vt: 6-8L/ kg (with ideal weight) = 500mL
RR: 12-20
PEEP: 0-5
FiO2: 100%
Flow: 40-60L/min

Tx of hyperventilation on AC mode of ventilation

-decrease RR
-decrease set Vt

SIMV

synchronized intermittent mandatory ventilation
-machine will recognize Pt breathing on own and the Vt that goes with it
-Pt is at risk for hypoventilation
-used for weening of Pt of vent.

SIMV Tx: hypoventilation

-back on AC

PC mode of ventilation

-press. control mode
-highest press: 30cm
-unnatural breathing
-unknown volume
-high sedation/ sometimes paralytics
concern:
-unknown TV=> increase CO2=> Resp. acidosis

APRV (airway pressure release ventilation)

provides 2 levels of CPAP (inspiration/ expiration)
*mode of ventilation along with spontaneous ventilation to promote lung recruitment of collapsed & poorly ventilated alveoli
*CPAP is released periodically for a brief period
*short realease along with spontaneous breathing promote CO2 elimination
*release time is short to prevent peak exp flow from returning to a zero baseline
-ARDS Pt
-best of AC/PC

CPAP/ BIPAP

-used for weening => longer intubated => longer BiPAP = greater than 2 weeks => trach.
-SAT: manage anxiety
includes:
-pressure support
-PEEP
-FiO2
RR/TV is Pt driven
RF: greater press => decreased CO

ARDS (acute respiratory distress syndrome)

respiratory insufficiency marked by progressive hypoxia
-3 stages: acute/ rebuild/ rehab
-“stiff-lung disease”
-decreased PaO2 despite increased FiO2 (refractory hypoxemia)
-decreased lung compliance
-no real Tx for ARDS
-Bed that pronates

ARDS s/s

-increased RR
-increased anxiety
-pulm. HTN
-Refactory hypoxemia
-decrease pulmonary compliance (elasticity)
-dyspnea
-noncardiac-associated bilateral pulmonary edema
-dense pulmonary infiltrates x-ray (ground-glass)

ARDS Treatment

-intubate w/ PEEP @ 15-18
-APRV
-Diuretics (non-cardiac origin edema/ crackles)
-supportive care (Remove causative factors)
-Pronation bed: definitive Tx chest lying/ drains lung system down and out
Administration of 100% oxygen

ARDS pathophysiology

Insult—activates the overwhelming inflammatory system (ex: PNA/ acute pancreatitis)

-systemic inflammatory response syndrome(SIRS)
-Release of inflammatory mediators
-Damage to alveolar-capillary membrane
-Increased capillary permeability and corresponding leakage of fluid
-Pulmonary edema (noncardiogenic)

Ventilator associated risks

-aspiration
-atrophy > 24 hours
-infection/ VAP > 48 hours = VAP induced
-barotrauma (Pneumothorax)
-unplanned extubation (Pt pulls out)

Extubation criteria

-reason for intubation resolved?
-stable ABG & acid-base
-mental status 0 to +1 on RASS
-airway positive/ patent (not swollen)
deflate & listen for air leak, you do not want to lose your airway if airway stays swollen

lungs response to hypoxia

-increased RR => resp. alk
-inflammation will cause:
edema
weepy membranes
decreased surfactant
increased WBC/ neutrophils
-pulmonary HTN

PE causes:

-non-movement/ bed-rest
-fat emboli
-DVT/ VTE
-amniotic fluid (pregnant)
-tumors
-septic vegatation (IV drug users)

PE patho

-clot formed and thrown to lung => pulmonary HTN in R. vent.

PE s/s

-hemoptosis
-agitation
-sob
-increased HR
-decreased BP (or unaffected)

PE diagnostic

-echo
-d-dimer assay (fragments/ positive thrombus formation/ also cancer-renal failure-infection-surgery)
-spiral CT w/ contrast
-Ventilation-perfusion scan (V/Q scan)
chest x-ray

PE Rx meds

-heparin therapeutic = 5,000 units
-heparin preventative = 8 units/ kg
monitor PTT/ aPTT/ activeXa
-TPA (breaks clot)
-Lovenox

examples of lipid tubing and how long are they good for before changing out?

-propofol
-TPN

Atropine info:

-anticholingeric drug: increase SA node automaticity and AV node conduction
-dilates pupils
-dry-up
-MOA: speed up HR
-IV admin
-assess for rise in HR

Atropine admin

-0.5 mg IV q 3-5 min
-max dose 3 mg

Atropine Tx

-symptomatic bradycardia
-asystole

Atropine nursing implication

-pupils
-may not work if the QRS is wide (ventricle)

Epinephrine class and effects

-catecholamine/ adrenergic
1. automaticity
2. contractility
3. vasoconstrictor (BP/ SVR increase)
-increase HR
-increase BG/ BS
-bronchodilator
1,2,3 reasons given first drug in “CODE” 1mg IVP q 3-5 min

Epinephrine used to treat

-asystole
-vtach(pulseless)
-v.fib
-PEA
-symptomatic bradycardia (IV infusion)

Amiodarone

-antidysrhythmic
given for:
-v-tach
-v-fib
-fast A-fib/ flutter
300 mg IVP/ 10 mins for CODE… 150mg all others

Amiodorone s/e

-lung tox
-photosensitivity
-hyper/hypo thyroid
-increased liver enzymes (ALT/ AST)
-neuro
-hypotension
-bradycardia

Amiodorone MOA

-decreased excitability
-prolongs action potential to terminate VT or VF

Amiodarone admin

-dead: 300 mg IV push followed by infusion at end of code
-alive: 150 mg IV bolus followed by infusion

Amiodarone Tx:

-dead v.tach
-v.fib
-fast v.tach
-a.fib
-a.flutter

Amiodarone nursing implications

-central line
-in-line filter
-long half-life
-cytochrome p450 enzyme
-monitor liver function

Amiodarone home teaching

-lung toxicity
-neuro toxicity
-thyroid toxicity
-wear sunscreen/ sunglasses

Magnesium

Tx: -torsades de pointe (caused by low Mg+)
-2 mg IV push if dead
-2 mg in 50-100mL over 60 mins if alive
nurse must monitor mag levels after admin

Adenosine

-MOA: slows conduction of AV node and interrupts AV nodal reentry circuits
-Admin: A/C IV site, 6 mg pushed rapidly followed by 20 mL NS, can repeat with 12mg
-Tx: fast atrial rhythms, slow down, may convert
-nursing implications: Pt may asystole for 15 seconds, educate Pt, chest pain, headache, if asthma: may complain of chest tightness/ bronchospasm

Dopamine

BP support
-dysrythmic
-use central line (if periph => necrosis)
-(alpha) increased BP (vasoconstriction & SVR)@ 10-20 mcg/kg/min
-(beta cells) increased contractility & CO @ 2-10 mcg/kg/min
-tachycardia

Dopamine s/e

-increased BP (10-20)
-increased contractility (2-10)
-*incrased HR = major => increases CO*
-great to use with bradycardic PT w/ atropine

Dopamine Tx

-hypotension
-symptomatic bradycardia
-occasionally for CO (not 1st line drug)

Dopamine nursing implications

-tachycardia
-increased dysrhythmias
-admin central line: extravasation => necrosis

Lidocaine class and s/e

Antidysrhythmic Meds
-numbness
-neuro tox (lighthead/drowsy)
-bradycardia
-hypotension (blocks)
-n/v
-constipation

V-tach stable: definition

-pulse: yes
-asymptomatic no Sx
150-100mg IVBolus/ 10 mins, then infusion of amiodorone

V-tach unstable: definition

-pulse : yes
-ALOC
-decrease BP
-“chest pain”
synchronous cardioversion
-then 150-100mg IVbolus/ 10 mins followed by infusion amiodorone
if torsades: magnesium instead of amiodorone

V-tach w/ no pulse

-CPR/ call for code “Help”
-De-fib when available
-CPR/ 1st drug Epi 1mg IVPush
-stop CPR/ analyze rhythm and De-fib
-resume CPR/ give 2nd drug amiodorone IVPush @ 300 mg
if torsades: magnesium instead of amiodorone

Cardiac dysrhythmias: unstable manifestations

-hypotension
-pale/ cool skin
-weakness
-angina
-dizziness/ syncope
-confusion/ disorientation
-SOB

Rapid response team call criteria

– >140/min HR <40/min
– >28 RR <8
-BP: SBP >180 or <90mmHg
-O2 sat <90%
-urine ouput <50mL over 4 hours (no Hx)
-staff concern

additional criteria:
-unrelieved chest pain from nitro
-threatened airway
-seizure
-uncontrolled pain

rapid response team:

RRT
-CCRN
-RT
-physician (critical or hospitalist)

work of breathing

the amount of energy needed to breath.

compliance lungs

define?

decrease this?

ease with which lungs and chest wall expand depends upon elasticity of lungs and surface tension

-ARDS, cystic fibrous

resistance lungs

things that block the flow of gas like bronchoconstriction, edema, mucous build up

poisition for airway control:

normal way?

neck injury?

head chin tilt lift

jaw thrust

devices to open airway:

OPA: prevents tongue from falling and blocking airway

NPO: you can suction through this

when to use OPA and NPO

for patients with decrease LOC and need an airway

indication for OPA

patient has nasal obstruction or epistaxis

indication for NPO

pt with bleeding disorder

how to confirm placement ETT, OPA, NPO?

-Xr chest should be 3-4 cm above carina

-Ausculatate both sides of chest, breath sounds bilateral and equal chest rise and fall

-listen to stomach, louder breath sounds will be heard over stomach than chest is placement wrong

indication for ventilation:

hypoxemia?


hypercapnia?

progressive deterioration?

-Ineffective oxygenation in blood. ABG: Pao2 <60 and on Fio2 >.50

Ineffective ventilation, high CO2. ABG: PCO2 >50 with pH <7.25

increase RR, decrease tidal volume, increase WOB

Tidal Volume (TV)

amount of air inhaled or exhaled with each breath under resting conditions

endotracheal intubation

define/preffered route?

used to do what?

-insertation of an endotracheal tube ETT through mouth or nose. Oral more common due to reduce infection risk

-maintain airway
-remove secretion
-prevent aspiration
-provide mechanical ventilation

patient needs artifical airway for longer than 10-14 days than they need what?

tracheostomy may be created to avoid mucosal and vocal cord damage due to ETT placement to long.

what must be by patient bed side at all times when they have ETT or tacheostomy?

ambu bag!!!

reduce leak for ETT regarding the cuff?

inflate the cuff until seal is made by having no harsh sounds heard over trachea when pt breaths in, but should have a slight air leak on peak inspiration.

is a patient able to talk with ETT?

no due to it goes into vocal cords and a proper seal would make it so they cant talk

ETT how much to inflate balloon?

20-30ml

extubation process of ETT?

-pre o2 and suction patient
-deflate cuff
-pt inhale at peak inspiration remove tube and suction airway
-after removal have them cough and deep breath to remove anything else

end tidal co2 detector?

used for ETT and is device that attaches to it.
-correct placement: carbon dioxide turns from purple to yellow
-not correct stays yellow

purple poopy
yellow good

nursing care for making sure ETT secure each shift?

-mark position through out shift
-skin assessment around mouth
-able to move but need 2nd RN or Resp therapist with you
-restain pt if needed

aseptic technique?

A procedure performed under sterile conditions.

ETT and trach suction:

type of technique?

how often to give oral care?

nursing care key points with suction?

-aseptic so sterile

-q2hours

-suction 3 passes no longer than 30 seconds in between
-suction on way coming out in rotation movement
-suction 10-15 suctions at a time

closed suction method?

catheter is in covered sheet so you can use it more than once. Dont need sterile clothes to suction this way

what to avoid when doing endotracheal suction?

Avoid normal saline installation into the tube

normal respiratory pressure?

positive pressure?

negative pressure chest rises and creates a negative pressure to suck air in.

-movement of gases into lungs through a positive pressure. this is like when patient is on ventilator

FiO2

define?

normal in air we breath?

fraction of inspired oxygen; the concentration of oxygen in the air we breathe

21% oxygen in air we breath

tidal volume:

define?

normal amount?

on ventilator adjust it to what?

amount of air we breath in during a normal breath

6-8mL/kg (ideal weight) about 500ml for adult patient

adjust it according to peak and plateau pressures

I:E ratio

Inspiratory/expiratory ratio; an expression for comparing the length of inspiration with that of expiration, normally 1:2, meaning that expiration is twice as long as inspiration (not measured in seconds)

ventilator setting four big ones?

-Fio2
-Tidal Volume
-Resp Rate
-I:E Ration
-PEEP

PEEP (positive end expiratory pressure)

define?

normal amount?

if to high setting on ventilatory what could happen?

times we want PEEP HIGH

gas pressure remaining in the system between breaths (during relaxation and before the next squeeze) this helps keep the alveoli open.

5-20cm H20

can cause reduced cardiac output and impede on venous return

-ARDS to keep fluid from building up

ETCO2 monitoring

-monitors what?

normal?

used when?

-alveolar CO2 as assess trends in patients ventilation status

30-43mm HG

-ETT, PCA, brain injury

Ventilator alarms:

KEY POINT WITH ALARMS?

three types?

NEVER turn off alarms

1. low pressure: low exhale volume, ETT disconnection or cuff leak, tube displacement

2. High pressure, excess secretion, kinks, bitting on tubes, bronchospasms, coughin

3. Apnea, no spontaneous breathing in period of time, used during whining time off ventilation

complications of mechanical ventilation: ETT malposition

define?

s/s?

how to check for this?

-This is when the ETT is placed to far in or into the right main stem bronchus

-unequal chest rise and fall, stomach moving during breathing if in esophagus

chest xray

complications of mechanical ventilation: unplanned Extubation

risk factors for this?

agitated, improper use of restraints

complications of mechanical ventilation: laryngeal and tracheal injury:

how does this occur?

damage during tube movement inside the mouth or high pressure of the balloon when inflated 25-35mm HG

complications of mechanical ventilation: mucosal damage:

occurs when?

tx?

tight tape, ETT pressing down on lip or tongue

-resposition tube and tape q24hours, assess skin

complications of mechanical ventilation: barotrauma:

what occurs during this?

s/s

tx?

to much PEEP and high tidal volume can cause pneumothorax

decrease breath sounds, decrease SPO2, sub q air around chest or neck

chest tube

complications of mechanical ventilation: oxygen toxicity

occurs when?

s/s?

FIO2 >50% and causes damage to the alveoli membrane leading to poor gas exchange

dyspnea, tachypnea, crackles, cyanosis in resp distress

ventilator associated infections:

big problem?

causes?

prevention?

pneumonia (VAP) very big problem.

-poor oral hygiene, aspiration of feeding, poor hand hygiene

lower sedation drugs, maintain HOB 30 at least, oral hygiene q2hours

complications of mechanical ventilation: stress ulcers/GI bleed

prevention?

-start tube feeding ASAP

-use H2 receptor blockers or PPI

-position changes

core ventilator bundle for prevention

-Head of bed 30 degrees

-Awaken daily and assess readiness to wean
-Stress ulcer prophylaxis

-DVT prophylaxis

-Oral care (chlorhexidine in some bundles)

questions to ask when determining if ready to wean off ventilator?

-is the underlying problem fixed?

-is patient stable: VS, EKG?

-pt able to follow commands

-pt able to cough, clear secretion

after ventilator wean off to what?

facemask or NC

when to stop the weaning process from a ventilator ?

-increase HR
-hypoxic
-unstable VS
-Restless
-Anxious

how to wean off ventilator for end of life care:

remove ETT and stop machine, provide comfort care

extubation assess for what?

-Stridor
-Hoarseness
-Change in Vital signs
-Low oxygen satuation

NPPV

noninvasive positive pressure ventilation this would be like CPAP or BIPAP

patient needs to do what after extubation?

cough, deep breath to clear secretion and use incentive spirometer

what can you use extubation period to prevent doing another ETT?

NPPV: noninvasive positive pressure ventilation this would be like CPAP or BIPAP

after extubation what most be done before having food?

swallow study before food or drinks

Noninvasive Positive-Pressure Ventilation (NPPV)

Used to prevent using invasive artificial airways (endotracheal [ET] tube or tracheostomy) in patients with acute respiratory failure, cardiogenic pulmonary edema, or exacerbation of chronic obstructive pulmonary disease. It has also been used following extubation of an ET tube.

Positive pressure ventilation PPV without artificial airway: includes?

1. Nasal Continuous Positive Airway Pressure (CPAP)
•Helps open airway open at sleep

2. Bi-level Continuous Airway Pressure (BiPAP)
•Two levels of pressure: higher with breath in, lower on out breathing
•COPD, prevent ETT

Noninvasive Positive-Pressure Ventilation (NPPV): CPAP or Bipap what are some requirements/contraindication?

ØTight seal
ØIntact respiratory drive
ØAble to protect airway

nursing care with NPPV?

-skin assessment around the mask
-FIO2 and pressure setting proper setting

tracheostomy:

indication?

types?

how much air to go in pilate balloon?

-ETT for 10-15 days and not recovering or in emergency setting where upper airway is occluded

-Cuff: unable to swallow and risk for aspiration use cuff to block that from occurring

-Without Cuff: use with patient able to swallow

20-25ml

tracheostomy:

complication?

-Fistula formation
-Trachea necrosis
-aspiration
-airway obstruction
-bleeding
-trachea stenosis

can anything besides air go into tracheostomy?

NO and to prevent this cover if needed in public but not to tight due to breathing through this

Tracheostomy obturator

goes in after hole is made to maintain size, than insert tube and remove this once tube is placed.

tracheostomy initial trach tie?

don’t remove for 24 hour due to is could close up

chest tube:

define?

three chambers in the chest tube machine?

tube into the pleural space to remove air or fluids

-first: collection
-second: water seal: acts like a one way valve, air coming out but no air back into patient
-third: control chamber

Chest tube management

Never clamp. Lower than the bed. Tape all connections. Look for bubbling in the chamber & tidaling with every breath.

chest tube: tidaling?

moving fluids in water chamber during resp pattern is normal

if no bubbling is occurring this means lung isn’t expanding or obstruction in system

Noncardiogenic pulmonary edema

“acute respiratory distress syndrome”, resulting from destruction of capillary beds that allows fluid to leak out, this allows a “WHITE out” on the chest Xray

ARDS pathophysiology

-Capillary endothelial cell damage
-Alveolar epithelial cell damage
-Increased vascular permeability
-Decreased production of surfactant
-Pulmonary edema and alveolar collapse

ARDS risk factors

-Sepsis (33% of cases)
-Aspiration of gastric contents
-Shock
-Infection
-Trauma
-Toxic inhalation
-Near drowning
-Multiple blood transfusions

ARDS S/S

-noncardiac-associated bilateral

-pulmonary edema; dense pulmonary infiltrates on x-ray (ground-glass appearance

-SOB, rapid shallow breathing, pale skin-blue, confusion, hypoxiemia

-Pao2<60 on ABG

-high pressure warning on ventilator due to alveoli collasping

-increased PIP on ventilation

Symptoms of ARDS

-Dyspnea and tachypnea

-Hyperventilation with normal breath sounds

-Respiratory alkalosis

-Increased temperature and pulse

-Worsening chest x-rays that progress to “white out”

-Increased PIP on ventilation

-Eventual severe hypoxemia

ARDS tx?

1. treat the cause
2. oxygenation and ventilation: PEEP help improve gas exchange, decrease oxygen consumption through pain control, sedation
3. comfort care: neuromuscular blocks to decrease o2 demand: Rocuronium

4. Position: prone/continous lateral rotation therapy

tx in ARDS prevent what complications?

-Ventilation pneumonia
-DVT
-stress ulcers
-nutrition issues
-tube placement confirm when moving

Status asthmaticus:

define?

nursing actions?

Life-threatening episode of airway obstruction that may be unresponsive to common treatment.

-prep ETT, IV fluids, o2, bronchodilators, epi, systemic steroid therapy

peak flow meter: colors?

Green is good

yellow: caution level use SABA “rescuer inhaler”

red: SAVA ASAP and hospital if no improvements

COPD:

define?

causes?

Chronic obstructive pulmonary disease (COPD) Irreversible disease causing hypoxemia and respiratory acidosis.

-tobacco smoke, dust, chemicals

informed consent?

full disclosure to patient or legal resp. possible risk and benefits of tx. On the doctor.

Advanced directives:

has the written wishes for the patients. Along with organ donations information should be in here.

Withholding life sustaining treatment: ?

withholding stopping the ventilator use, comfort measures are still done for the patient

CPR decisions:

client and family has right to when they want it or not. AMA support family from stopping CPR.

Client autonomy

pt makes their own decisions. If impaired by sedation than find next of Kin

end of life care decisions must be made by who?

next of kin or power of attorney

behavioral pain scale:

used when?

includes what?

scoring?

-widely used in the ICU setting and was developed specifically to assess pain in the mechanically ventilated patient.

The behavioral pain scale score includes facial expressions, limb movements, and compliance with ventilation.

3-12

regional anesthesia:

two types?

-spinal anesthesia: injection of local anesthetic into CSF creates autonomic, sensory and motor blockade

epidural anesthesia: local anesthetic into epidural space, doesnt enter CSF, low dose: sensory high dose: sensory and motor

spinal anesthesia:

patho?

injection of local anesthesia into CSF in the subarachnoid space usually below L2. since going into CSF goes body wide creating autonomic, sensory and motor blockade

autonomic: vasodilation and low blood pressure

motor block: cant move

sensory: no pain

epidural anesthesia:

patho?

local anesthetic into epidural space via thoracic or lumbar approach. doesnt enter CSF but binds to nerve roots in spinal cord.

low dose: sensory blocked but motor intact

high dose: sensory and motor blocked

epidural anesthesia:

how its given?

-catheter is left in place so more doses can be given PRN

regional anesthesia:

desired effects?

vasodilation, analgesia, better surgical outcomes

spinal vs epidural anesthesia:

which one more rapid?

duration?

spinal onset more rapid due to going into CSF

both can be extended in duration time by using indwelling catheters to allow additional doses

regional anesthesia: spinal and epidural:

complication?

autonomic nervous system blocks can cause:
-low BP, low HR, N/V

less with epidural vs spinal

other:

postdural puncture headahces

back pain

isolated nerve injury

meingitis

regional anesthesia: spinal and epidural:


how do you know if dose to high?

patient may feeling tingling in arms and hands along with inadequate breathing and apnea

General anesthesia drugs:

Barbiturates: Methohexital

advantage:?

adverse effects?

nursing action?

-rapid induction, duration <5min

-myocardial depression, low BP, RR depression, involuntary movements

-tx nausea

General anesthesia drugs:

Nonbarbiturate: Etomidate:

advantages?

useful with type of patient?

adverse effects?

nursing actions?

-produces little change in cardiac health, little RR impact, no histamine release

-hemodynamically unstable patients

-myoclonia, N/V, hiccups, adrenocortical inhibition

-assess for transient skeletal muscles movements- myoclonia, N/V, low BP, low BG

General anesthesia drugs:

Nonbarbiturate: Propofol:

ideal for type of patient?

white color from?

side effects?

nursing action

-short outpatient procedures because rapid onset and metabolic clearance. but also used for induction and maintenance of anesthesia

lipid base so gives it creamy white color

-low HR, dysrhythmias, low BP, apnea, transient phlebitis, hypertriglyceridemia “hyperlipidemia”

-monitor for HR, BP, triglyceride q24hours when sedated >24hours

General anesthesia drugs:

inhalation agents volatile liquids: Isoflurane, desflurane, sevoflurane

MOA of all of them?

Isoflurane- key points?

desflurane- Key points?

Sevoflurane- key points?

skeletal muscle relaxation

no increase ventricular irritability, no liver or renal toxicity

fastest onset, and emergence, least post op cognitive issues, possible airway irritant

-less impact on heart and RR, rapid acting. Preferred for inhalation induction as non irritation to resp tract

General anesthesia drugs:

inhalation agents volatile liquids: Isoflurane, desflurane, sevoflurane

side effects?

which ones not for CAD patients?

which one for emergence delirium tx?

-RR depression, low BP, myocardial depression, seizure

Isoflurane and Desflurane

sevoflurane

General anesthesia drugs:

inhalation agents: gaseous agents like nitrous oxide:

MOA?

used when?

potentiates volatile agents thus speeding induction and reducing total dose needed and side effects

weak anesthetics, so its rarely used by self usually used with volatile agents (Isoflurane, desflurane, sevoflurane).

General anesthesia drugs:

inhalation agents: gaseous agents like nitrous oxide:

must be given how?

contraindication?

with o2 to prevent hypoxemia

-avoid patient with strong hx of N/V and bone marrow depression

General anesthesia drugs: Dissociative anesthetic like Ketamine


MOA?

side effects?

tx for hallucinations, agitation?

analgesic and amnesic “loss of memory”

-hallucinations and nightmare, ICP, IOP, increase HR/BP

benzo

adjuncts to general anesthesia:

opioids: fentanyl, morphine, methadone

MOA?

side effects?

induce and maintain anaesthesia, reduce stimuli from sensory nerve ending, provide analgesia during surgery and recovery

-RUSH: resp depression, urinary retention, sedation, low BP ADD on: peripheral vasodilation when combined with anesthetics, pruritus

adjuncts to general anesthesia:

benzos: midazolam, diazepam, lorazepam:

MOA?

careful with what other drug?

reverse drug?

reduce anxiety, induce and maintain anesthesia, induce amnesia (loss of memory), treat emergence delirium, supplement regional anesthesia

-synergistic effect with opioids increasing potential for RR depression, low BP, sedation and confusion

flumazenil

adjuncts to general anesthesia:

neuromuscular blocking agents: Depolarizing agents like Succinylcholine:

good for what?

side effects?

-ETT, by promoting skeletal muscle relaxation (paralysis)

Resp muscle paralysis, confusion, N/V, weakness

adjuncts to general anesthesia:

neuromuscular blocking agents: Depolarizing agents like Succinylcholine:

if intubated monitor?

reverse drug?

return of muscle strength, LOC, ventilation

maintain patients airway until able to cough and return muscle strength

neostigmine (prostigmin)

adjuncts to general anesthesia:

nondepolarizing agents: Pancuronium, rocuronium

reverse of these drug?

anticholinesterase- neostigmin, pyridostigmine

adjuncts to general anesthesia: antiemetics

examples?

side effects?

zofran, reglan, scopolamine (patch)

Headache, dizziness, IV irritation, dysrhythmias, dysphoria, dystonia, dry mouth, central nervous system sedation.

adjuncts to general anesthesia: Dexmedetomidine:

used when?

side effects?

induces and maintains sedation in nonETT patient prior to and/or during surgical procedures

-low BP, low HR, sinus arrest, transient HTN during loading dose

adjuncts to general anesthesia: dexamethasone:

used when?

counteracts emetic effects of inhalation agents and opioids

palliative care:

define?

goals?

form of care and tx that focuses on reducing severity of disease and symptoms

prevent and relieve suffering, and improve quality of life for patients with life limiting illness

hospice define?

main goal?

includes?

for patients with 6months or less to live

assist patient live fully as comfortable

symptoms management, advance care planning, spiritual care, family support

end of life care:

define?

final phase of patients illness when death is imminent

death rattle:

define

causes

noisy wet sounding RR

caused by mouth breathing and build up of mucous in airway

cheyne stroke define

alternative periods of apnea and deap rapid breathing

Kubler-Ross stages of grief

1. Denial
2. Anger
3. Bargaining
4. Depression
5. Acceptance

The Grief Wheel

normal

loss

1. Shock: numbness, denial, disbelief

2. protest. anger, guilt, sadness

3. disorganization: anxiety, apathy, confusion

4. reorganization: return to normal

new normal

resp center location?

regulated by what?

pon and medulla of the brain stem

chemoreceptors in the reso center, aorta, carotid arteries. Sense increase levels of Co2, low level of o2 and send impulse to spinal cord to phrenic nerve to breath

work of breathing?

amount of energy needed to breath.

disease lung patient need more energy to breath resulting in use of accessory muscles. Leads to becoming tired and may need mechanical ventilation help to breath

compliance:

define?

decrease when?

increased when?

measures how well the lungs can stretch

decrease with lung diseases like pulmonary fibrosis, ARDS, pulmonary edema. This results in lungs becoming stiff and need more energy to breath.

asthma and emphysema which means lungs over stretch due to loss of elasticity resulting in air being trapped.

resistance in Resp system:

define?

increase when?

opposition to flow of gases

-restricted airway, bronchoconstriction, increased mucus or increased edema.

how can compliance and resistance be measured?

spirometer

normal pH

7.35-7.45

The respiratory system will compensate for a metabolic problem (kidneys) how fast does this occur?

vs

The metabolic system (kidneys) will compensate for a respiratory problem—compensation can take how long?

compensation occurs rapidly.

a few days to occur

PaCO2:

considered what?

connection to patient?

an acid so if it is elevated that means body has more acid. If lower than body has less acid.

reflection of how well the patient is ventilating.

hyperventilation- PaCo2 is low due to breathing fast greater amount of co2 leaving the body

hypoventilation: paCo2 is high due to patient not breathing off co2 and holding more inside

Hco3:

considered what?

a base so if elevated body has more base and if lower it has less of a base

normal ABG value:

pH: 7.35-7.45
PaCO2: 35-45
Hco3: 22-26
PaO2: 80-100

Ph: 7.30
PaCO2: 50
HCO3: 27
PaO2: 60

?

This is a partially compensated respiratory acidosis. The pH is low and the PaCO2 is elevated. It is partially compensated because the HCO3 is now abnormal. This means that the kidneys are beginning to retain more bicarbonate. It is not enough bicarbonate because the pH is still abnormal.

Ph: 7.36
PaCO2: 31
HCO3: 18
PaO2: 85

?

The answer is fully compensated metabolic acidosis. The pH is 7.36 which is normal but leans towards the acidotic side. The PaCO2 is low. A low PaCO2 is alkalosis which does not match the pH. The PaO2 is normal which indicates that it is not a respiratory problem. The HCO3 is low which is acidotic. Therefore it is a metabolic acidosis.

hypoxia:?

hypoxemia?

early signs of poor oxygenation?

late signs?

not enough oxygen

-not enough oxygen in the blood

EARLY: RAT:
Restless, anxious, tachycardia, pale skin, elevated BP, nasal flaring

LATE to BED: Bradycardia, extreme anxious, dyspnea, confusion, low BP, cyanotic skin, resp acidosis

resp failure:

causes?

-Resp causes:
ARDS, asthma, pneumonia, COPD, pulmonary edema, pulmonary emboli, pneumonthorax, pulmonary HTN


nonpulmonary: mental status changes, head injury, drug OD, spinal cord injury, hemorrhage, MSK injury, HR

resp failure treated with?

oxygen
ventilation
prevention of complication

nasal cannula:

amount of oxygen?

nurse considerations?

1L to 6L/min (24-44% FiO2)

-use water soluble gel to prevent dry nares
-humidification for 4L or more

simple face mask:

amount of oxygen?

nursing consideration?

5L-8L (40-60% FiO2)

monitor skin breakdown
-teach pt wear mask
-nasal cannula when eating
-flow rates less than 5 can result in rebreathing of co2

venturi mask:

amount of o2?

nursing consideration?

4-10 L/min (24-50% FiO2)

-perfect for COPD
-simple face mask with adapter
-most precise o2 concentration

partial non rebreathing mask:

amount of oxygen?

nursing consideration

6-11 (40-75% FiO2)

-reservoir bag with no value allows client to rebreathe up to one third of exhaled air together with room air.
-Keep bag from deflating by adjusting oxygen rate
-assess for skin break down

non rebreather mask

amount of o2?

nursing consideration

10-15l/min (60-80% FiO2)

allow bag to fill up

-oneway valve allows pt to inhale max o2 from bag.
-two exhalation ports have flaps with prevent room air from coming in
-hourly assessment of valves and flaps

Aerosol and Humidity Delivery Systems

oxygen amount?

for what type of patients?

At least 10 L/min ( 24-100 FiO)

-humidity face mask for patient without articifal airway

-good for pt that cant tolerate mask- facial trauma, burns, thick secretion

Aerosol and Humidity Delivery Systems

type for trach patient?

type for ETT patient?

key point?

Trach collar

T- piece

-dont cover the T piece, and make sure nothing pulling it

Manual Resuscitation Bag (aka ambu bag)

amount of o2?

15L/min

oxygen toxicity:

define?

s/s?

nursing action?

results from high concentration of oxygen (above 50%), long duration of oxygen therapy (24-48hours) and the degree of lung disease

-nonproductive cough
-subternal pain
-nasal stuffiness
-N/V, fatigue, H/A

-lower o2 amount to maintain normal spo2, ABG monitor. use CPAP or biPAP to help decrease amount of oxygen needed, PEEP if on mechanical ventilator to decrease need of oxygen

acute respiratory failure:


define?

results when one of both o2 and co2 is impaired. as in insufficent o2 transferred to blood to insufficent co2 removed from the lungs

hypercapnic respiratory failure

INCREASED CO2 (greater than 45mm Hg) and DECREASED pH (less than 7.35), Telling us it’s a VENTILATION problem, pt either not breathing enough or has an airtrapping diagnosis such as asthma or COPD

hypoxemic respiratory failure

define?

causes?

Low PaO2 with low or normal PaCO2, SaO2 <90% despite FiO2 >0.6


Caused from disease of the lung
V/Q mismatch and intrapulmonary shunting are the major pathophysiology mechanisms

respiratory failure:

classifed by what?

levels for each one?

hypoxemic: or oxygenation failure due to not enough o2 transfer between alveoli and pulmonary capillaries.
-Pao2<60 on >60% o2

hypercapnic or ventilatory failure because issue removing co2.
-PaCO2 >45 and pH <7.35

four causes for hypoxemia and hypoxemia resp failure?


two most common?

1. mismatch between ventilation V and perfusion Q. “V/Q mistmatch”

2. Shunting

3. Diffusion limitation

4. Alveolar hypoventilation


most common: V/Q mismatch and shunting

V/Q mismatch

define?

increased examples?

An imbalance in the amount of oxygen received in the alveoli and the amount of blood flowing through the alveolar capillaries

-COPD, pnuemonia, asthma where increase secretion are present in airway which creates imbalance

A shunt

occurs when?

two types?

occurs when blood leavers the heart without getting gas exchange

1.anatomic: ventriocular septal defect

2. Intrapulmonary: blood flows through pulmonary capillaries without getting gas exchange. occurs during ARDS,

Alveolar hypoventilation

define?

examples

A generalized decrease in ventilation that results in an increase in the PaCO2 and a consequent decrease in PaO2.

-chest wall injury, asthma, neuromusclar disease

hypoxemia? vs hypoxia?

when amount of o2 in arterial blood is low

when Pao2 falls and can lead to hypoxemia

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